What is quercetin? Quercetin's effect on the lungs

What is quercetin? Quercetin's effect on the lungs

Quercetin, also known as quercetin or quercetin, is a natural flavonoid discovered in 1936 by Hungarian physiologist Szent Gyorgyi. It is widely present in the stem skin, flowers, leaves, 

buds, seeds and fruits of many plants, especially in the stem and leaves of buckwheat, sea buckthorn, hawthorn, onion and other plants. Quercetin has a variety of biological activities,

including antioxidant, anti-inflammatory, immunomodulatory properties, which make it in the field of medicine and health care has a wide range of application prospects.


Quercetin's effect on the lungs

Quercetin has potential benefits for lung health in the following ways:

Antioxidant effect:

Quercetin neutralizes free radicals and reduces oxidative stress, thereby protecting lung cells from damage. This has important implications for the prevention

and treatment of lung diseases caused by oxidative stress.

Anti-inflammatory effect:

Quercetin has been shown to inhibit the release of several pro-inflammatory molecules, such as tumor necrosis factor-α, interleukin-6, and so on, thereby reducing

the inflammatory response. This has a positive effect on alleviating chronic lung inflammation, such as asthma and chronic obstructive pulmonary disease.

Immunoregulatory effects:

Quercetin can enhance the body's immune function and improve the ability to fight infection. This is essential for fighting respiratory infections and maintaining healthy lungs.

To improve emphysema:

Studies have shown that quercetin can inhibit emphysema caused by dust gases such as cigarettes and smoke. It can reduce airway cell inflow, oxidative damage and inflammatory factor levels,

 improve lung tissue structure, and thus reduce the degree of emphysema. Therefore, quercetin can be used as an adjunct treatment for emphysema,

 but it needs to be used rationally under the guidance of a doctor.

Other protective effects:

Quercetin can also play a protective role in the lungs through other mechanisms, such as inhibiting fibroblast proliferation, reducing collagen synthesis, and reducing the risk of pulmonary fibrosis.

 

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